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3 Result(s) for ' DEXA'
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1.
Anti-Obesity Effects of Cissus Quadrangularis (Cissuslean®) in C57bl/6j Mice with High-Fat-Diet Induced Obesity and 3t3-L1 Adipocytes
Sudhanva M S, Akhina Tom, Firoz H M, Zayeem Firoz Hussain, Sadashiva CT, Shobith Rangappa
Journal of Food and Nutrition Research. 2025 13 (1). doi: 10.12691/jfnr-13-1-2
Keywords: Anti-obesity,Cissus quadrangularis,Cissuslean®, DEXA ,high-fat diet
Context: Obesity is gradually becoming a widespread health problem, and treatment using natural compounds has seen an increasing trend. The aim of this study investigated the effects and underlying mechanisms of Cissus quadrangularis (Cissuslean®) on adipogenesis and high-fat diet (HFD)-induced obesity. The inhibitory effects of Cissuslean® on adipocyte differentiation were evaluated in vitro using 3T3-L1 preadipocyte cells, focusing on AMPK signaling and lipid metabolism. Subsequently, an in vivo trial with male C57BL/6J mice fed HFD was conducted to examine the effects of Cissuslean® at 25, 50, and 100 mg/kg on adipose tissue characteristics, body weight, fat mass, and obesity-related gene expression. Cissuslean® significantly inhibited preadipocyte differentiation by reducing intracellular lipid accumulation and suppressing the expression of key adipogenic regulators. It also enhanced AMPK phosphorylation during early adipocyte differentiation. In HFD-induced obese mice, Cissuslean® administration resulted in marked reductions in body weight gain, adipose tissue mass, and hepatic lipid accumulation. Furthermore, decreased serum ALT and AST levels indicated attenuation of HFD-induced hepatic injury. According to these results, Cissuslean® reduces body fat mass by inhibiting lipase activity and regulating genes and proteins involved in adipogenesis and lipogenesis in epididymal adipose tissue. Our results suggest that Cissuslean® may be used as an anti-obesity agent.
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2.
Cushing Syndrome Due to Pigmented Nodular Adrenal Hyperplasia in an Iranian Patient: A Case Report and Review of the Literature
Kamyar Asadipooya, Roohollah Narimani, Ashraf Aminorroaya, Bijan Iraj‬, Mohammadreza Kalantarhormozi, Novin Afshar Moghadam, Elaheh Kavoussi, Mitra Dashtizadeh‬‬‬
American Journal of Medical Case Reports. 2021 9 (10). doi: 10.12691/ajmcr-9-10-3
Keywords: Cushing syndrome, PPNAD, Primary pigmented nodular adrenocortical, hirsutism, low dose DEXA methasone suppression test
Context: Primary pigmented nodular adrenocortical disease (PPNAD) is a rare cause of Adrenocorticotropic hormone-independent Cushing syndrome. In this case, we present a case of PPNAD in 25 y/o women who presented with a 3 years history of weight gain, hirsutism, and abnormal menstruation. Our findings were suggestive of PPNAD in the pathological study of adrenal biopsy.
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3.
Maturation of the Thalidomystery
Yossi Cohen, Odit Gutwein, Osnat Garach-Jehoshua, Adina Bar-Chaim, Abraham Kornberg
International Journal of Hematological Disorders. 2014 1 (1A). doi: 10.12691/ijhd-1-1A-1
Keywords: Thalidomide, lenalidomide, DEXA methasone, gene expression profile, PLSCR1, differentiation
Context: Despite years of investigations, the mechanism of action of thalidomide and its derivatives is still unsolved. Recently we elaborated on the basis of the current literature data, including x-ray images of thalidomide victims, a hypothesis that premature differentiation of developing limb elements underlies the teratogenic effects of thalidomide and may also account for the antitumoral activity of the IMiDs. To examine the feasibility of this theory, we searched within gene expression profile datasets, which analyzed the changes induced by the IMiDs in myeloma cells, for supportive evidence. As expected, our analysis revealed a reproducible upregulation of a cluster of differentiating genes induced after either in-vivo or in-vitro treatment of the tumor-cells with thalidomide/ lenalidomide. This finding debates the long standing dogma that ascribed the teratogenic effect of thalidomide to apoptosis or toxic injury to limb bud mesenchyme/blood-vessels and places the IMiDs in line with differentiating teratogens like retinoids and related compounds.
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