Figure 4. PU inhibits ER stress-associated liptoxicity in response to PA exposure: In the presence or absence of PU, cells were exposed to PA for 24 h, and western blotting was used to analyze the key ER stress markers including GRP78, the phosphorylation of PERK, IRE1α and eIF2α, as well as the cleaved-PARP

From

Punicalagin Inhibits Palmitate Acid-induced Lipoapoptosis through Inhibition of ER Stress, and Activation of SIRT1/Autophagy in HepG2 Cells

Han Zhang, Hao Wu, Shutao Yin, Hongbo Hu, Chong Zhao

Journal of Food and Nutrition Research. 2020, 8(9), 536-542 doi:10.12691/jfnr-8-9-10