Figure 4. E-OJ modulates MAPK signaling pathways in HeLa cells. (A) HeLa cells were treated with 10 μg/ml of E-OJ for the indicated time points, and total and phosphorylated forms of p38, JNK, and ERK1/2 were analyzed by Western blotting. (B) ERK1/2 activation was quantified as the ratio of phospho-ERK1/2 to total ERK1/2. (C) Cells were exposed to E-OJ (5, 7.5, or 10 μg/ml), kaempferol (K), quercetin (Q), or gallic acid (G) for 15 minutes, and ERK1/2 phosphorylation was assessed. GAPDH served as a loading control. Data were obtained from three independent experiments. *p < 0.05; **p < 0.01; ***p < 0.001

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Orostachys Japonicus Promotes Apoptosis in Cervical Cancer Cells through Modulation of NF-ΚB and ERK1/2 Pathways

Seon-Hee Kim, Dong Seok Lee

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